Collie is generally resilient and healthy, but there are some health problems that can affect the breed. Not all diseases are caused solely by genetic factors, but environmental factors also play a major role. One of the major culprits are overweight or very underweight. The use of BMI (Body Mass Index) can be a good guide if you are unsure of the dog's weight by feeling the dogs back and sides or watching the dog according to a Body Condition Score Chart. Distribution of nutrition must also be in proper balance and is in proportion to the dog's energy consumption.

Collie Eye Anomaly (CEA), a genetic disorder that causes improper development of the eye and possible blindness in the worst case, there is a common ailment in the breed.

CEA can occur in two forms:

  • Chorio-Retinal Dysplasia (CH)
  • Colobom (optic nerve or tendon membrane hernia hernia)

Chorio-Retinal Dysplasia are underdeveloped in the choroid and the overlying retina in an area in the back of the eye, near synsnervens indmunding that can cause loss of vision, but 80-90% of the dogs have no vision problems. In approx. 30% of cases can facilitate change "disappear" again. This is called "go normal" phenomenon. That's because growing pigment-containing cells over the affected area, which is hereby covered, when the puppy gets older. Therefore, puppies should be examined from 6 to 10 weeks of age. A recent large Swedish study (over 8000 Collier) has shown that about. 70% had CH, which also corresponds to the Danish Kennel Club registrations for 2009.

Colobom is a more comprehensive error development of the sclera with the missing parts of the sclera and therefore leads to a "hernia" through which the retina and blood vessels are squeezed out. They speak of a partial or total optic nerve hernia, and the disease can cause more serious complications, bleeding in the eye, retinal detachment and blindness. The disease occurs fortunately not as frequent. In Denmark it is estimated that around. 5% of Collies and Shetland Sheepdog is hit.

Through genetic testing and careful screening program, it would be theoretically possible to eliminate both these problems in the breed lines. Some people claim the problem is exacerbated by less strict breeding standard introduction, but there are no scientific studies to support this. Collie puppies should be screened at an early age by a certified veterinary ophthalmologist to check for both of these eye problems.

More rarely, Collies be affected by progressive retinal atrophy (PRA), another genetic disease of bilateral degeneration of the retina lead to progressive vision loss culminating in blindness. Retinal Dysplasia (RD) is a faulty development of the retina in fetuses and can cause blindness. In 2009 only had very few cases of RD. Persistent pupilmembran (PPM) Persistent hyperplastic Tunica Vasculosa Lentis and Persistent hyperplastic primary Vitreus PHTVL / PHPV is also more rarely occurring eye diseases collie.

Canine cyclic neutropenia is a cyclic blood disease that is usually fatal to affected puppies. The disease is also called "gray collie syndrome" due to hit the puppy with a pale gray, pink / gray or beige colors, none of which are normal collie colors. The dogs have brown or pale sable noses, but never black noses, which they should have. Puppies through adulthood plagued by immune diseases throughout their lives, and rarely live more than three years and often die a few weeks after birth. DNA testing may help detect carriers of the recessive gene that causes the disease.

Hip dysplasia (HD) is as with most of the major races, a potential concern for collies. HD is thought to have a polygenetisk multifactorial background. It is believed that there are multiple genetic causes, each of which can be influenced by various "non-hereditary" conditions. These conditions are, for example. overfeeding, the size, growth rate, hormonal factors muscles mm. But how does this "cause cocktail" exactly the composition is not known. We know today that there is a direct correlation between 1øshedsgraden in the hip joint and the development of clinical HD. Ie the longer the rod ends can be moved out of the socket, the greater is the risk of developing HD. It is assumed that puppies are born with normally developed hip joints, and then early in their lives to develop the unstable hip joint caused by hereditary and the aforementioned environmental factors (feeding, exercise, growth rate, etc.).. Feeding is the best documented environmental factor for the development of HD. Several well-documented studies show that over-feeding with similar obesity increases the severity of HD. Overfeeding with lime or a wrong relation between calcium and phosphorus in the diet also increases the degree of HD. Dogs with an inherited propensity to simultaneously fed, has a very high risk of developing HD. Over Exercise or failure loads (slippery floors, prolonged violent play) of the puppy's hip condition worse if there is a tendency to instability in the hips. The disease usually occurs in two scenarios. One occurs when the dog is 5-12 months and manifested by severe pain, the puppy often balk at exercise or in severe lameness. The second scenario occurs later in the dog's life (often after 4 years) and have a more chronic development. It manifests itself in a milder degree of pain, but will worsen with the dog's age, when it first started.

The table shows the registered collie dog HD status for 2009.

reg. i 2009 Status Status description
80% A A free of dysplasia
7% B in transition between free and mild dysplasia
4% C mild dysplasia
7% D moderately severe dysplasia
2% E severe dysplasia

Multi-Drug-Sensitivity (MDR1) is the abbreviated name of a gene called multi-drug resistance first A mutation in this gene results in sensitivity to ivermectin and a number of other substances. Dogs with the mutation will respond to these drugs. Having two copies of the mutation will lead to side effects, but with a single copy can also provide some sensitivity to certain drugs. Dogs with this mutation have a transport error - the substance that goes into their minds, fail to be transported out, and builds up to toxic levels. This causes serious neurological problems including seizures and sometimes death. The defective gene is present in several breeds but are well known among collies.